Sunday, April 21, 2013

Celeste H Nettles - Psychology Today

[unable to retrieve full-text content]I also work with clients experiencing relationship challenges as well as mental health concerns such as mood disorders, ADHD, and anxiety. I help individuals to recover from traumatic events in their lives and restore their self ...

Source: http://therapists.psychologytoday.com/rms/152122

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Friday, April 19, 2013

Knee bracing can 'significantly' reduce pain of kneecap osteoarthritis

Knee bracing can 'significantly' reduce pain of kneecap osteoarthritis [ Back to EurekAlert! ] Public release date: 19-Apr-2013
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Contact: Alison Barbuti
alison.barbuti@manchester.ac.uk
44-016-127-58383
University of Manchester

Wearing a knee brace has been shown to 'significantly improve the pain and symptoms' of a type of osteoarthritis affecting the kneecap, according to a new study

Wearing a knee brace has been shown to "significantly improve the pain and symptoms" of a type of osteoarthritis affecting the kneecap, according to a new study.

Arthritis Research UK-funded researchers at The University of Manchester claim their findings, presented at the Osteoarthritis Research Society International meeting in Philadelphia tomorrow (Friday April 19) have enormous potential for treating this common joint condition effectively as well as providing a simple and cheap alternative to painkillers.

Osteoarthritis of the knee affects around six million people in the UK and is increasing as the population ages and becomes more obese. Current treatments are limited to pain relief and joint replacement.

Osteoarthritis of the knee affecting the kneecap (patellofemoral osteoarthritis) accounts for about 20% of patients with knee pain. They typically experience pain that is made worse by going up and down stairs, kneeling, squatting and prolonged sitting.

"There's a pressing need for non-surgical interventions for knee osteoarthritis, and little attention has been paid to treatments particularly aimed at the kneecap (the patellofemoral joint), a major source of knee pain," explained Dr Michael Callaghan, research associate in rehabilitation science at the University of Manchester.

"We've shown that something as simple as a lightweight knee brace can dramatically improve the symptoms and function for people with this particular type of knee osteoarthritis."

The research team conducted a randomised controlled trial of a lightweight lycra flexible knee brace fitted around the knee with a support strap for the kneecap. One hundred and 26 patients between the ages of 40 and 70 were treated over a 12-week period. All had suffered from arthritic knee pain for the previous three months.

They were randomly allocated to either immediate brace treatment or delayed treatment (i.e. after six weeks.) Both groups of patients eventually wore the brace for a period of 12 weeks and averaged roughly seven hours a day.

After six weeks of brace wearing there were significant improvements between the brace wearing group and the no treatment group in scores for pain, symptoms, knee stiffness, muscle strength and function. After 12 weeks there were significant improvements in these scores for all patients compared to when they started.

"Patients repeatedly told us that wearing the brace made their knee feel more secure, stable, and supported," Dr Callaghan added. "Our theory is that these sensations gave the patient confidence to move the knee more normally and this helped in improving muscle strength, knee function and symptoms."

Professor Alan Silman, medical director of Arthritis Research UK, which funded the trial, said: "Osteoarthritis of the knee is a painful disorder that affects millions of people in the UK, causing pain and reducing activities. We know that in patients with arthritis, the knee joint is frequently out of normal alignment, which might be an underlying cause of the problem, as well as making it worse.

"By using a simple brace, the researchers have been able not only to correct the alignment but achieve a very worthwhile benefit in terms of reducing pain and function. This approach is a real advance over relying on pain killers and has the potential to reduce the end for joint surgery and replacement, procedures often employed when the symptoms become uncontrollable."

###

The ROAM (Research into Osteoarthritis in Manchester) project has run three trials at The University of Manchester and the University of Salford. The project is led by internationally renowned Boston-based osteoarthritis expert Professor David Felson, with funding of 1.8m from Arthritis Research UK.


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AAAS and EurekAlert! are not responsible for the accuracy of news releases posted to EurekAlert! by contributing institutions or for the use of any information through the EurekAlert! system.


Knee bracing can 'significantly' reduce pain of kneecap osteoarthritis [ Back to EurekAlert! ] Public release date: 19-Apr-2013
[ | E-mail | Share Share ]

Contact: Alison Barbuti
alison.barbuti@manchester.ac.uk
44-016-127-58383
University of Manchester

Wearing a knee brace has been shown to 'significantly improve the pain and symptoms' of a type of osteoarthritis affecting the kneecap, according to a new study

Wearing a knee brace has been shown to "significantly improve the pain and symptoms" of a type of osteoarthritis affecting the kneecap, according to a new study.

Arthritis Research UK-funded researchers at The University of Manchester claim their findings, presented at the Osteoarthritis Research Society International meeting in Philadelphia tomorrow (Friday April 19) have enormous potential for treating this common joint condition effectively as well as providing a simple and cheap alternative to painkillers.

Osteoarthritis of the knee affects around six million people in the UK and is increasing as the population ages and becomes more obese. Current treatments are limited to pain relief and joint replacement.

Osteoarthritis of the knee affecting the kneecap (patellofemoral osteoarthritis) accounts for about 20% of patients with knee pain. They typically experience pain that is made worse by going up and down stairs, kneeling, squatting and prolonged sitting.

"There's a pressing need for non-surgical interventions for knee osteoarthritis, and little attention has been paid to treatments particularly aimed at the kneecap (the patellofemoral joint), a major source of knee pain," explained Dr Michael Callaghan, research associate in rehabilitation science at the University of Manchester.

"We've shown that something as simple as a lightweight knee brace can dramatically improve the symptoms and function for people with this particular type of knee osteoarthritis."

The research team conducted a randomised controlled trial of a lightweight lycra flexible knee brace fitted around the knee with a support strap for the kneecap. One hundred and 26 patients between the ages of 40 and 70 were treated over a 12-week period. All had suffered from arthritic knee pain for the previous three months.

They were randomly allocated to either immediate brace treatment or delayed treatment (i.e. after six weeks.) Both groups of patients eventually wore the brace for a period of 12 weeks and averaged roughly seven hours a day.

After six weeks of brace wearing there were significant improvements between the brace wearing group and the no treatment group in scores for pain, symptoms, knee stiffness, muscle strength and function. After 12 weeks there were significant improvements in these scores for all patients compared to when they started.

"Patients repeatedly told us that wearing the brace made their knee feel more secure, stable, and supported," Dr Callaghan added. "Our theory is that these sensations gave the patient confidence to move the knee more normally and this helped in improving muscle strength, knee function and symptoms."

Professor Alan Silman, medical director of Arthritis Research UK, which funded the trial, said: "Osteoarthritis of the knee is a painful disorder that affects millions of people in the UK, causing pain and reducing activities. We know that in patients with arthritis, the knee joint is frequently out of normal alignment, which might be an underlying cause of the problem, as well as making it worse.

"By using a simple brace, the researchers have been able not only to correct the alignment but achieve a very worthwhile benefit in terms of reducing pain and function. This approach is a real advance over relying on pain killers and has the potential to reduce the end for joint surgery and replacement, procedures often employed when the symptoms become uncontrollable."

###

The ROAM (Research into Osteoarthritis in Manchester) project has run three trials at The University of Manchester and the University of Salford. The project is led by internationally renowned Boston-based osteoarthritis expert Professor David Felson, with funding of 1.8m from Arthritis Research UK.


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?


AAAS and EurekAlert! are not responsible for the accuracy of news releases posted to EurekAlert! by contributing institutions or for the use of any information through the EurekAlert! system.


Source: http://www.eurekalert.org/pub_releases/2013-04/uom-kbc041913.php

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Sunday, April 7, 2013

Ford EcoSport Gets Soft Launch in Kochi

[unable to retrieve full-text content]

Source: www.ibtimes.com --- Saturday, April 06, 2013
Ford's much anticipated EcoSport SUV has got a soft launch in Kochi, Kerala as part of company's 12-city campaign road show. ...

Source: http://www.ibtimes.comhttp:0//www.ibtimes.co.in/articles/454281/20130406/ford-ecosport-suv-india-launch-preview-price.htm

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Different drug combinations work best for prevention versus treatment of colorectal tumors

Different drug combinations work best for prevention versus treatment of colorectal tumors [ Back to EurekAlert! ] Public release date: 7-Apr-2013
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Contact: Diana Quattrone
diana.quattrone@fccc.edu
215-728-7784
Fox Chase Cancer Center

Study evaluates the effectiveness of a non-steroidal anti-inflammatory drug and the cholesterol-lowering medication Lipitor in animal models more relevant to humans

WASHINGTON, DC (April 7, 2013)Colorectal cancer is the second leading cause of cancer-related deaths in the United States. Once colorectal cancer has spread to other parts of the body, only 11 percent of patients will survive five years from the date of their diagnosis. Most colorectal cancers are adenocarcinomascancers that begin in cells that make and release mucus and other fluids. Adenocarcinomas begin as benign tumors called adenomas, which become malignant over time. By treating adenomas before they become cancerous, it could be possible to prevent colorectal cancer.

Researchers at Fox Chase Cancer Center have tested the effectiveness of two promising drugs in preventing and treating colorectal adenomas in mice. A team led by Wen-Chi Chang, PhD, assistant research professor at Fox Chase, found that the effect of these drugs, which have already been approved by the Food and Drug Administration for the treatment of other conditions, depends on whether adenomas are present when drug treatment begins. Chang will present these findings at the AACR Annual Meeting 2013 on Sunday, April 7.

"We often get focused on either the preventive or therapeutic setting and don't think about how these drugs are maybe serving more than one purpose," says senior author on the study Margie L. Clapper, PhD, co-leader of the Cancer Prevention and Control Program at Fox Chase. "The most exciting thing for us was to be able to track these tumors and for the first time distinguish between prevention and chemotherapy, and to show that one agent is maybe effective in both settings if used appropriately, or in this case, in combination with another agent."

Past studies in animals have shown that colorectal tumors can be suppressed by combined treatment with two drugs: a non-steroidal anti-inflammatory compound called sulindac and a cholesterol-lowering medication called atorvastatinwhose brand name is Lipitor. But in those studies, tumors were induced in an unnatural waythrough exposure to carcinogenic chemicalswhereas in humans, cancer often has genetic origins.

To evaluate the effectiveness of sulindac and atorvastatin in an animal model more relevant to humans, Chang, Clapper and their colleagues used a unique mouse that had genetic alterations that cause them to develop multiple colorectal adenomas, without exposure to carcinogens. "No one had previously tested the effectiveness of this drug combination against colorectal cancer originating from alterations in the genome," Clapper says. "In some ways, using this type of preclinical tumor model represents a new paradigm for doing prevention studies and therapeutic studies."

In the new study, the researchers treated the mice with either drug alone or in combination for 100 days and used colonoscopic examinations to evaluate the presence and size of tumors before and after treatment. In mice that had tumors prior to treatment, only combination therapy reduced the number of adenomas in the colon by the end of the treatment period.

The results were strikingly different in mice that were tumor-free when treatment began. In these mice, exposure to atorvastatin alone or in combination with sulindac resulted in about a three-fold increase in the percentage of mice that were tumor-free by the end of the treatment period. Among these mice, 44 percent of those treated with atorvastatin alone and 30 percent of those treated with both drugs did not develop tumors, compared with 13 percent of mice that received no treatment and nine percent that received sulindac alone. Moreover, atorvastatin treatment completely inhibited the formation of microscopic adenomas in these mice.

The findings demonstrate that the effectiveness of the two drugs at preventing and treating colorectal adenomas depends on whether tumors are present prior to the onset of treatment. "Based on this study, we're able to say that if you don't have a tumor to begin with, maybe Lipitor is best, but if you do have a tumor to begin with, you need the combination therapy," Chang says. "We can start to tailor clinical care based upon the disease state as well as the establishment of tumors."

Moving forward, the researchers plan to study the specific genetic alterations in this particular mouse model, with the goal of identifying molecular pathways that could be targeted with therapies.

###

Co-authors on the study include Christina M. Ferrara, Stacy L. Mosier, Harry S. Cooper, Karthik Devarajan, Harvey Hensley, and Tianyu Li of Fox Chase. This research was supported by NIH R21CA129467.

Fox Chase Cancer Center, part of Temple University Health System, is one of the leading cancer research and treatment centers in the United States. Founded in 1904 in Philadelphia as one of the nation's first cancer hospitals, Fox Chase also was among the first institutions to receive the National Cancer Institute's prestigious comprehensive cancer center designation in 1974. Fox Chase researchers have won the highest awards in their fields, including two Nobel Prizes. Fox Chase physicians are routinely recognized in national rankings, and the Center's nursing program has achieved Magnet status for excellence three consecutive times. Fox Chase conducts a broad array of nationally competitive basic, translational, and clinical research and oversees programs in cancer prevention, detection, survivorship, and community outreach. For more information, call 1-888-FOX-CHASE (1-888-369-2427) or visit http://www.foxchase.org.


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AAAS and EurekAlert! are not responsible for the accuracy of news releases posted to EurekAlert! by contributing institutions or for the use of any information through the EurekAlert! system.


Different drug combinations work best for prevention versus treatment of colorectal tumors [ Back to EurekAlert! ] Public release date: 7-Apr-2013
[ | E-mail | Share Share ]

Contact: Diana Quattrone
diana.quattrone@fccc.edu
215-728-7784
Fox Chase Cancer Center

Study evaluates the effectiveness of a non-steroidal anti-inflammatory drug and the cholesterol-lowering medication Lipitor in animal models more relevant to humans

WASHINGTON, DC (April 7, 2013)Colorectal cancer is the second leading cause of cancer-related deaths in the United States. Once colorectal cancer has spread to other parts of the body, only 11 percent of patients will survive five years from the date of their diagnosis. Most colorectal cancers are adenocarcinomascancers that begin in cells that make and release mucus and other fluids. Adenocarcinomas begin as benign tumors called adenomas, which become malignant over time. By treating adenomas before they become cancerous, it could be possible to prevent colorectal cancer.

Researchers at Fox Chase Cancer Center have tested the effectiveness of two promising drugs in preventing and treating colorectal adenomas in mice. A team led by Wen-Chi Chang, PhD, assistant research professor at Fox Chase, found that the effect of these drugs, which have already been approved by the Food and Drug Administration for the treatment of other conditions, depends on whether adenomas are present when drug treatment begins. Chang will present these findings at the AACR Annual Meeting 2013 on Sunday, April 7.

"We often get focused on either the preventive or therapeutic setting and don't think about how these drugs are maybe serving more than one purpose," says senior author on the study Margie L. Clapper, PhD, co-leader of the Cancer Prevention and Control Program at Fox Chase. "The most exciting thing for us was to be able to track these tumors and for the first time distinguish between prevention and chemotherapy, and to show that one agent is maybe effective in both settings if used appropriately, or in this case, in combination with another agent."

Past studies in animals have shown that colorectal tumors can be suppressed by combined treatment with two drugs: a non-steroidal anti-inflammatory compound called sulindac and a cholesterol-lowering medication called atorvastatinwhose brand name is Lipitor. But in those studies, tumors were induced in an unnatural waythrough exposure to carcinogenic chemicalswhereas in humans, cancer often has genetic origins.

To evaluate the effectiveness of sulindac and atorvastatin in an animal model more relevant to humans, Chang, Clapper and their colleagues used a unique mouse that had genetic alterations that cause them to develop multiple colorectal adenomas, without exposure to carcinogens. "No one had previously tested the effectiveness of this drug combination against colorectal cancer originating from alterations in the genome," Clapper says. "In some ways, using this type of preclinical tumor model represents a new paradigm for doing prevention studies and therapeutic studies."

In the new study, the researchers treated the mice with either drug alone or in combination for 100 days and used colonoscopic examinations to evaluate the presence and size of tumors before and after treatment. In mice that had tumors prior to treatment, only combination therapy reduced the number of adenomas in the colon by the end of the treatment period.

The results were strikingly different in mice that were tumor-free when treatment began. In these mice, exposure to atorvastatin alone or in combination with sulindac resulted in about a three-fold increase in the percentage of mice that were tumor-free by the end of the treatment period. Among these mice, 44 percent of those treated with atorvastatin alone and 30 percent of those treated with both drugs did not develop tumors, compared with 13 percent of mice that received no treatment and nine percent that received sulindac alone. Moreover, atorvastatin treatment completely inhibited the formation of microscopic adenomas in these mice.

The findings demonstrate that the effectiveness of the two drugs at preventing and treating colorectal adenomas depends on whether tumors are present prior to the onset of treatment. "Based on this study, we're able to say that if you don't have a tumor to begin with, maybe Lipitor is best, but if you do have a tumor to begin with, you need the combination therapy," Chang says. "We can start to tailor clinical care based upon the disease state as well as the establishment of tumors."

Moving forward, the researchers plan to study the specific genetic alterations in this particular mouse model, with the goal of identifying molecular pathways that could be targeted with therapies.

###

Co-authors on the study include Christina M. Ferrara, Stacy L. Mosier, Harry S. Cooper, Karthik Devarajan, Harvey Hensley, and Tianyu Li of Fox Chase. This research was supported by NIH R21CA129467.

Fox Chase Cancer Center, part of Temple University Health System, is one of the leading cancer research and treatment centers in the United States. Founded in 1904 in Philadelphia as one of the nation's first cancer hospitals, Fox Chase also was among the first institutions to receive the National Cancer Institute's prestigious comprehensive cancer center designation in 1974. Fox Chase researchers have won the highest awards in their fields, including two Nobel Prizes. Fox Chase physicians are routinely recognized in national rankings, and the Center's nursing program has achieved Magnet status for excellence three consecutive times. Fox Chase conducts a broad array of nationally competitive basic, translational, and clinical research and oversees programs in cancer prevention, detection, survivorship, and community outreach. For more information, call 1-888-FOX-CHASE (1-888-369-2427) or visit http://www.foxchase.org.


[ Back to EurekAlert! ] [ | E-mail | Share Share ]

?


AAAS and EurekAlert! are not responsible for the accuracy of news releases posted to EurekAlert! by contributing institutions or for the use of any information through the EurekAlert! system.


Source: http://www.eurekalert.org/pub_releases/2013-04/fccc-dd040213.php

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Thursday, March 28, 2013

Award-winning screenwriter Fay Kanin dies at 95

FILE - In this March 3, 2006 file photo, former president of the Academy of Motion Picture Arts and Sciences, Fay Kanin, poses for a photo during the foreign language film award reception at the Academy of Motion Picture Arts and Sciences, in Beverly Hills, Calif. The Academy confirmed Kanin?s death Wednesday, March 27, 2013. She was 95. The Emmy-winning and Oscar-nominated screenwriter served as president of the film academy from 1979 to 1983. (AP Photo/Danny Moloshok, File)

FILE - In this March 3, 2006 file photo, former president of the Academy of Motion Picture Arts and Sciences, Fay Kanin, poses for a photo during the foreign language film award reception at the Academy of Motion Picture Arts and Sciences, in Beverly Hills, Calif. The Academy confirmed Kanin?s death Wednesday, March 27, 2013. She was 95. The Emmy-winning and Oscar-nominated screenwriter served as president of the film academy from 1979 to 1983. (AP Photo/Danny Moloshok, File)

(AP) ? Emmy-winning and Oscar-nominated screenwriter Fay Kanin has died. She was 95.

The Academy of Motion Picture Arts and Sciences confirmed Kanin's death Wednesday. She served as president of the film academy from 1979 to 1983.

Kanin was nominated for an Academy Award for 1958's "Teacher's Pet" alongside her husband and writing partner, Michael Kanin. The film starred Clark Gable and Doris Day.

Fay Kanin was also recognized for her television contributions, winning two screenwriting Emmys in 1974 and another for producing the TV special "Friendly Fire" in 1979.

Details on Kanin's survivors and cause of death were not immediately available.

Associated Press

Source: http://hosted2.ap.org/APDEFAULT/4e67281c3f754d0696fbfdee0f3f1469/Article_2013-03-27-US-Obit-Fay-Kanin-/id-4012a784a4df4e0daad1da24dabd227b

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Wednesday, March 27, 2013

NYC aquarium rebounds, rebuilds after Sandy

NEW YORK (AP) ? The New York Aquarium has cherished its big-city setting by the sea for half a century. But the ocean that is the aquarium's lifeblood dealt it a shattering blow last fall.

Superstorm Sandy's surge overran carefully calibrated tanks with oily, debris-filled water, knocked out even backup power to all the exhibits and made it impossible to check on some of them for days. Managers contemplated shipping animals away and wondered whether the institution itself could survive in its spot on Coney Island.

Five months later, more than 80 percent of the collection is intact, and visitors should be able to see walruses, angelfish, otters and others when about half the aquarium reopens late spring. A planned expansion remains on track, now coupled with rebuilding and floodproofing an institution that aims to be an object lesson in enduring on the shore.

"I don't think we could abandon this facility. Not that we didn't think about it ? we thought through everything," aquarium Director Jon Forrest Dohlin said this week as he stood amid pipes and cables in a now-empty jellyfish exhibit.

"We want to be here, and we also want to be able to talk to the community about what we did, how we handled this, and how the city of New York can start to look toward the future of living in this coastal environment."

As he walked through the 14-acre grounds, penguins watched like squat sentries from their outdoor habitat. Walruses snoozed as sea lions arced through the air on their trainers' cues, staying in practice for shows to resume in a few months. Angelfish and other tropical species shimmered around a coral reef and hefty pacu, a fruit-eating piranha relative, hovered in an Amazonian display in the one building where exhibit space wasn't flooded.

But the effects of the Oct. 29 storm were still starkly visible elsewhere.

The floor was torn out of a building that houses jellyfish, seahorses, lungfish and other unusual creatures. Many were still there but set to start moving next month to other aquariums while their facility is rebuilt. The open pool in front of it was drained dry; it housed hundreds of freshwater koi that died in the saltwater surge.

Sharks, sea turtles and rays circled serenely in a tank in the aquarium's veterinary hospital. They're healthy but were shuttled there after the storm put an exclamation point on plans to reinvent their exhibit. Nearby, the gutted cafeteria still has "Happy Halloween!" signs on its windows.

There's no firm date yet for this spring's partial reopening. The rest of the exhibits, including the new $120 million shark display, are to open in 2016.

Meanwhile, the Wildlife Conservation Society, which runs the aquarium, is determining how much insurance and government aid may pay toward fixing roughly $65 million in estimated damage.

The aquarium was founded in 1896 in lower Manhattan. It moved in 1957 to Coney Island, a faded seaside playground now striving for rebirth. Drawing more than 750,000 visitors a year, it's "the economic engine for Coney Island," says City Councilman Domenic Recchia Jr., who represents the area.

Aquariums are often built by the water and have proven vulnerable to hurricanes. New Orleans' Audubon Aquarium of the Americas lost thousands of fish when generators failed after Hurricane Katrina in 2005. It reopened about five months later.

In Galveston, Texas, Hurricane Ike's storm surge in 2008 killed about three-quarters of the fish in Moody Gardens' rainforest exhibit, General Manager Robert Callies said. The exhibit reopened in 2011 after bringing back hundreds of birds, reptiles and mammals sent to other zoos after the storm.

At the New York Aquarium, Sandy's surge coursed through air-intake vents in flood doors under the Coney Island boardwalk, punched through sand into the parking lot and rushed in from the parking lot after a creek overflowed blocks away.

As the water rose three feet high in Dohlin's ground-floor office, he watched it pour down a stairwell into a basement that housed exhibits and the equipment that keeps them alive.

"'We lost the aquarium,'" he thought.

Basements were under up to 15 feet of water. Generators were either damaged or useless because equipment needed to distribute their power was fried. The pump house that draws from the ocean to refresh the 1.5 million-gallon exhibits was out of commission, as were systems that treat the seawater, tailor it to different environments and maintain the oxygen levels, temperatures and water chemistry the aquarium's 12,000 animals need.

None had been evacuated. That would have been very difficult to arrange in the few days the aquarium had to prepare, Dohlin said.

Scrambling to save the collection, 18 staffers used hospital-style canisters to get crucial oxygen into the water, rebuilt filters and pumps on the fly and called in equipment from the Wildlife Conservation Society's four zoos. They mixed artificial seawater in garbage cans and warmed rooms with space heaters to keep water temperatures up, animal operations director David DeNardo said.

At the same time, managers weighed how much longer they had to get systems going before having to ship animals away, an unwelcome prospect for already stressed creatures. On Nov. 1, the wildlife society announced that a decision would probably have to be made in 24 hours. But key systems were at least partially running in all the exhibits two days later, and the animals stayed.

The koi and some other fish were dead. But many other fish and all the mammals were fine ? including Mitik, an orphaned walrus calf that arrived only weeks before. He seemed to enjoy splashing in a couple of feet of surge water, Dohlin said.

A 3-foot-long American eel disappeared from its tank but turned up, unharmed, in a staff shower stall. Seahorses held on to life despite the cold, dirty surge water that flowed into their tropical tanks.

Now, plans call for raising the new shark building several feet higher to meet new flood-zone predictions, moving air intake vents from the flood doors to the roof, moving electrical panels out of basements and installing full-height storm doors on some glass doors that were only partly protected.

It's an unexpected chance, Dohlin says, to improve both the aquarium's exhibits and endurance at once.

"Not to let any crisis go to waste," he said. "That's the real opportunity here."

___

Follow Jennifer Peltz at http://twitter.com/jennpeltz

Source: http://news.yahoo.com/nyc-aquarium-rebounds-rebuilds-sandy-065224810--finance.html

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Monday, March 4, 2013

NASA: SpaceX Dragon capsule to reach ISS on March 3rd at 6:01AM ET

Didn't get enough of the Dragon capsule launch this week? Good news, because after a day's delay due to (now remedied, according to NASA and SpaceX) faults with three clusters of its Draco thrusters, the capsule is set to be grappled by the International Space Station at 6:01AM ET on March 3rd (tomorrow morning). If you'll recall, the mission is mainly aimed at getting refreshed supplies and some experiments up to the space station. As an aside, NASA also notes that Dragon is still set to arrive back on earth for a splashdown on the 25th, as initially planned. If you're up for it, NASA TV coverage starts at 3AM the same day and the final berthing process (actually getting the capsule connected to the ISS) should happen at about 7:30AM -- all that said, initial "orbital manuevers" are set for 2AM, according to a tweet from Elon Musk. For more details on this stage of the mission, including those involved, blast over to the NASA source link below -- and make sure you've got enough coffee ready.

Filed under: , ,

Comments

Via: The Verge

Source: NASA, SpaceX (Twitter), Elon Musk (Twitter)

Source: http://www.engadget.com/2013/03/02/nasa-spacex-dragon-capsule-to-reach-iss-on-march-3rd-at-6-01-et/

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